Publié par happy-diet mercredi 13 janvier 2010

Syndrome have been described as acute respiratory failure in 1967 was accompanied by severe deficiency oxygenation and pulmonary double leakages and decreased plasticity, pulmonary

And usually occurs without the scourge of pulmonary precedent.

Invited on behalf of the acute respiratory distress syndrome in adults (ARDS) and the taking occurred since that is growing. Intensive care and was able to improve the

Survival rate after infection, but nevertheless still high mortality is unacceptable.

Pathological physiology

Physiological disorder lies in the acute respiratory distress syndrome when the veteran is on the rise outside the vascular fluid in the lungs. And as they are known,

Movement of fluid in the lung vascular permeability controlled by the balance between static pressure Liquid Hydrostatic Pressure and pressure solution can

Oncotic Pressure powers static Hydrostatic Forces encourages infiltration of liquids, while pressure solution can encourage Arthaffha. In the case

Natural forces are in control of the nomination and go down the movement of fluid from the blood to the interstitial spaces, however, and the fluid does not accumulate

In the interstitial spaces due to the effectiveness of the lymphatic vessels that remove fluid accumulated in the interstitial spaces and return to the general circulation. The

After all, the ability of the lymphatic vessels remain limited, and if exceeded the infiltration capacity of fluids to the lymphatic vessels Nzhaa accumulated fluid in

Interstitial spaces.

This accumulation is the initiation of the interstitial tissue on the broad road and air pulmonary arteries and later in the follicular walls (edema matrix),

Which leads to a decrease in flexibility with pulmonary slip, but rarely lead to the disruption of blood gases. But if development continues, the increased fluid

The accumulation affects vesicular spaces and the resulting two consequences:

1 - Climate liposome surface forces leading to a decrease in respiratory plasticity and low lung volumes.
2 - Flood impede vesicular lung ventilation leading to the issuance of the object Pettmasha without blood purification and to occur within the pulmonary Ext

lntrapulmonary Shunt, which is why the most important deficiency in severe hypoxia, which characterizes respiratory distress syndrome when the veteran.

That those changes in two Starling relationship is of great clinical importance. Patients living with Cardiogenic pulmonary edema have a pressure

Poetic static high. And the fluid leachate generally poor in protein content, and the liquid is a great picture nomination.

While in the respiratory distress syndrome when the veteran static capillary pressure is usually normal and the primary disorder leading to accumulation of fluid

Is a disorder of alveolar permeability in the diaphragm which is caused by capillary damage in the lining of the vascular and epithelial vesicular.

The causes of victimization in some cases very clear Kmat lung or inhalation of viral infectious fluids, but in most cases ambiguous due

To the immunologic mechanism or biochemical intervene to activate complement, and acid metabolites Aloracedoni, and leukocytes and release of root oxygen.

All this leads to edema and matrix vesicular fluids high protein content similar to those in the catalog. While we do not have changed the pressure

Solution can is the most important reason in the incidence of lung edema and the decrease in pressure within the vascular solution can increase the speed of fluid nominated in both the cases of edema

Pulmonary high-pressure and low pressure. In contrast, any increase in static pressure and poetic that triggers an increase in capillary permeability

Raising the speed nominated liquids.

In addition to the obvious victimization lung, the respiratory distress syndrome when the veteran partnering with other systemic manifestations. These functional changes

Outside the lung is often difficult to differentiate them from the troubles caused by hemorrhage that led to respiratory distress syndrome when the veteran. Apart

On the pathogen, some of these changes be descriptive of the respiratory distress syndrome in a veteran to the point with them and later on part of an entity

Syndrome procedure. And more descriptive of these changes is the lack of ability to increase tissue oxygen extraction in the face of lack of oxygen supply to them. It

The second is for the lack of a multi-functional, progressing gradually. Each disorder is another warning sign of bad.

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